Zie ook de literatuurlijst niet-toxische middelen en behandelingen specifiek bij darmkanker van arts-bioloog drs. Engelbert Valstar 

31 maart 2022: Bron: Journal of Clinical Oncology

Uit een subgroep van de 701 deelnemende patiënten uit de Maya studie bij zwaar voorbehandelde patiënten met uitgezaaide darmkanker en geselecteerd op microsatellietstabiel (MSS) en geen actieve O6-methylguanine-DNA methyltransferase (MGMT) blijkt dat als deze patiénten na twee kuren met temolozomide - temodal stabiele ziekte hebben en geen progressie van deze ziekte en vervolgens de immuuntherapie combinatie krijgen de mediane ziekteprogressietijd en mediane overall overleving verbeterde. De mediane ziekteprogressie vrije tijd en de totale overleving waren respectievelijk 7,0 en 18,4 maanden en het totale responspercentage was 45%.

Probleem is wel dat van de patiënten die door de eerste selectie kwamen er maar weinig toekwamen aan de immuuntherapie. De onderzoekers schrijven ook nadrukkelijk dat vervolgstudies verder onderzoek waard is maar wel vooraf via biomoleculair onderzoek patiënten met uitgezaaide darmkanker beter selecteren op temozolomide-/dacarbazine-gevoelige tumoren.

Hier hoe de patiënten in de Maya studie zijn geselecteerd op verschillende momenten:

Figure

Vertaald uit het abstract:

Van de 716 vooraf gescreende patiënten kwamen 204 patiënten (29%) moleculair in aanmerking en 135 patiënten begonnen aan het eerste behandelingsgedeelte. Hiervan werd bij 102 patiënten (76%) de behandeling stopgezet vanwege overlijden of ziekteprogressie na eerste kuren met temozolomide, terwijl 33 patiënten (24%) die ziektecontrole bereikten, begonnen met het tweede behandelingsgedeelte en de laatste onderzoekspopulatie vertegenwoordigden.

Na een mediane follow-up van 23,1 maanden (interkwartielbereik 14,9-24,6 maanden), was het PFS-percentage van 8 maanden 36%. De mediane PFS en de totale overleving waren respectievelijk 7,0 en 18,4 maanden en het totale responspercentage was 45%. Graad 3-4 immuungerelateerde bijwerkingen waren huiduitslag (6%), colitis (3%) en hypofysitis (3%). Er werden geen onverwachte bijwerkingen of aan de behandeling gerelateerde sterfgevallen gemeld.

Het volledige studierapport is gratis in te zien of te downloaden. Klik daarvoor op de titel van het abstract:

This is a multicenter, single-arm phase II trial evaluating the efficacy and safety of an immune-sensitizing strategy with temozolomide priming followed by a combination of low-dose ipilimumab and nivolumab in patients with microsatellite-stable (MSS) and O6-methylguanine–DNA methyltransferase (MGMT)–silenced metastatic colorectal cancer (mCRC).

Patients with pretreated mCRC were centrally prescreened for MSS status and MGMT silencing (ie, lack of MGMT expression by immunohistochemistry plus MGMT methylation by pyrosequencing). Eligible patients received two priming cycles of oral temozolomide 150 mg/sqm once daily, days 1-5, once every 4 weeks (first treatment part) followed, in absence of progression, by its combination with ipilimumab 1 mg/kg once every 8 weeks and nivolumab 480 mg once every 4 weeks (second treatment part). The primary end point was the 8-month progression-free survival (PFS) rate calculated from enrollment in patients who started the second treatment part, with ≥ 4 out of 27 subjects progression-free by the 8-month time point as decision rule.

Among 716 prescreened patients, 204 (29%) were molecularly eligible and 135 started the first treatment part. Among these, 102 (76%) were discontinued because of death or disease progression on temozolomide priming, whereas 33 patients (24%) who achieved disease control started the second treatment part and represented the final study population. After a median follow-up of 23.1 months (interquartile range, 14.9-24.6 months), 8-month PFS rate was 36%. Median PFS and overall survival were 7.0 and 18.4 months, respectively, and overall response rate was 45%. Grade 3-4 immune-related adverse events were skin rash (6%), colitis (3%), and hypophysitis (3%). No unexpected adverse events or treatment-related deaths were reported.

The MAYA study provided proof-of-concept that a sequence of temozolomide priming followed by a combination of low-dose ipilimumab and nivolumab may induce durable clinical benefit in MSS and MGMT-silenced mCRC.

© 2022 by American Society of Clinical Oncology
CONTEXT

  • Key Objective

  • The MAYA trial was designed to prospectively provide clinical proof-of-concept of the role of temozolomide as an immune-sensitizing agent because of induction of hypermutation in proficient mismatch repair/microsatellite-stable, O6-methylguanine–DNA methyltransferase–silenced metastatic colorectal cancer.

  • Knowledge Generated

  • The MAYA strategy allowed to achieve durable disease control to low-dose ipilimumab and nivolumab combination in heavily pretreated patients, along with a manageable safety profile.

  • Relevance

  • The MAYA strategy is worth further investigation in properly selected patients with metastatic colorectal cancer and may be agnostically translated to other temozolomide-/dacarbazine-sensitive cancers.

PRIOR PRESENTATION

Presented at the Colorectal Cancer Proffered Paper Session at the European Society for Medical Oncology 2021 Congress, Paris, France, September 18, 2021.

SUPPORT

Partial financial support and ipilimumab/nivolumab supply by Bristol Myers Squibb; temozolomide supply by Sun Pharma Italia.

CLINICAL TRIAL INFORMATION

NCT03832621

Conception and design: Federica Morano, Filippo Pagani, Filippo de Braud, Filippo Pietrantonio

Financial support: Filippo Pietrantonio

Provision of study material or patients: Francesca Bergamo, Maria Alessandra Calegari, Elena Conca, Maria Di Bartolomeo

Collection and assembly of data: Federica Morano, Alessandra Raimondi, Filippo Pagani, Sara Lonardi, Lisa Salvatore, Chiara Cremolini, Sabina Murgioni, Federica Palermo, Lorenzo Antonuzzo, Nicoletta Pella, Patrizia Racca, Michele Prisciandaro, Monica Niger, Francesca Corti, Francesca Bergamo, Alberto Zaniboni, Margherita Ratti, Michele Palazzo, Celeste Cagnazzo, Maria Alessandra Calegari, Federica Marmorino, Silvia Brich, Elena Tamborini, Federica Perrone, Maria Di Bartolomeo, Filippo de Braud, Filippo Pietrantonio

Data analysis and interpretation: Federica Morano, Alessandra Raimondi, Giovanni Randon, Michele Prisciandaro, Francesca Corti, Iolanda Capone, Elena Conca, Adele Busico, Massimo Di Maio, Massimo Milione, Filippo Pietrantonio

Manuscript writing: All authors

Final approval of manuscript: All authors

Accountable for all aspects of the work: All authors

The following represents disclosure information provided by authors of this manuscript. All relationships are considered compensated unless otherwise noted. Relationships are self-held unless noted. I = Immediate Family Member, Inst = My Institution. Relationships may not relate to the subject matter of this manuscript. For more information about ASCO's conflict of interest policy, please refer to www.asco.org/rwc or ascopubs.org/jco/authors/author-center.

Open Payments is a public database containing information reported by companies about payments made to US-licensed physicians (Open Payments).


Consulting or Advisory Role: Amgen, Servier, MSD Oncology, Merck

Research Funding: Bristol Myers Squibb (Inst), Astrazeneca (Inst)

No other potential conflicts of interest were reported.

ACKNOWLEDGMENT

The authors thank the patients and their families and caregivers for their participation.

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